Platismatia lacunosa
Crinkled rag lichen
Why is she so shape? I don't know. One of the many perplexing mysteries of the lichen kingdom.
images: source
Y'all, I just won €400 in a Science Slam competition that is going toward my research, and I want to thank all of you for being my audience for the last few years. Without this space where I can practice my science communication and my bad lichen jokes, I wouldn't have done nearly as well. Much love to you!
See me here looking extremely uncomfortable to be perceived by others
Lactose: e-excuse me...
Stomach: hey, what do we have here? Some goddamned lactose!
Small intestine: we don't like lactose here, ya know? You're gatecrashing a very private party!
Large intestine: (pushes him, making him stumble) there's two ways you can leave here... The easy way, or the hard way. What's it gonna be?
Lactose: but I...
Lactase: (deep voice) step back, everyone. (walks up and puts his arm around lactose's shoulders) he's with *me*
Large intestine: lactase?!
Small intestine: b-but you're both...
Lactase: I *said* he's with me. You got a problem with that?
Stomach: (finishes sizing him up) right. Course not. It's cool, lactose. Just don't cause any *problems*, you hear?
Lactose: I-I w-
Lactase: you don't have to answer that, babe. Just keep walking
Plant Immune System Part 3
The plant immune system is the topic of my PhD thesis, which I'm currently writing following several years of lab-based research as a PhD student at Imperial College London under the supervision of Professor Colin Turnbull.
Here's an introduction to my research, which focused on how certain plants defend themselves against aphids.
Aphids are an important insect pest that threaten agriculture worldwide. As we learned in the previous post, plant resistance (R) genes control resistance to specific pests and pathogens through interaction with effectors from the invaders. Since examples of R gene-dependent aphid resistance have been documented in different plant species, aphid-specific R genes may enable the development of resistant crops.
In the model plant Medicago truncatula, there are some varieties that are resistant to aphids and other varieties that are susceptible to Pea Aphids (Acyrthosiphon pisum). Whether the plant is resistant also depends on the variety of aphid. In my project, the A17 plant is resistant to PS01 aphids but not to N116 aphids, while the DZA plant is susceptible to both aphid varieties.
What is the key difference in the resistant versus susceptible plants? Resistant A17 plants have a portion of their genome “Resistance to Acyrthosiphon pisum 1” (RAP1) which determines resistance to PS01 aphids, but the genes controlling the defence response and physiological defence mechanisms remain unknown. Two candidate R genes located in RAP1, designated “RAP1A” and “RAP1B”, may control resistance.
My main objective in my PhD project has been to determine whether RAP1A and RAP1B control aphid resistance, and to investigate the RAP1-mediated defence response. I look forward to sharing the findings in publications and in talks next year!
Image credit: Original diagram by Katia Hougaard with images from the Turnbull Lab.
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Scientist, scholar, hapless train wreck all wrapped into one neurotic package.
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